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| # | Term | Definition | From Set |
|---|---|---|---|
| 1 | Glucocorticoids | One of the major groups of steroid hormones produced by the adrenal cortex. It enables the body to resist long-term stressors, primarily by incresing blood glucose levels. | Anatomy Lab I- Endocrine Glands |
| 2 | Glucocorticoids | Inflammation, Others | Pharmacology Test 2 multiple choice |
| 3 | glucocorticoids | steroid hormone | ch. 9 anatomical terms |
| 4 | Glucocorticoids | One of the major groups of steroid hormones produced by the adrenal cortex. It enables the body to resist long-term stressors, primarily by incresing blood glucose levels. | endocrine system |
| 5 | glucocorticoids | raises blood sugar levels | AP Bio Terms |
| 6 | Glucocorticoids | Hormones produced by the adrenal cortex designed to help the body fight stress | Homeostasis/Endocrine |
| 7 | glucocorticoids | target organ: liver, cause liver to make glucose from fats and proteins and to release new glucose into blood, also targets other cells to use fats for fuel instead of glucose, also strong anti-inflammatory agents, EX: cortisol | Nehah's Biology Vocab |
| 8 | Glucocorticoids | zona fasciculata of cortex of adrenal gland | 211 Lab Test 1 |
| 9 | Glucocorticoids | inhibit phospholipase A2- inhibits arachidonic acid and production of leukotrienes, cytokines and prostaglandins (also interfere with transcription factors required to activate T cell genes) | Immunomodulators |
| 10 | Glucocorticoids | one of the two main corticosteroids from adrenal cortex; increases energy supplies during stress by promoting synthesis of glucose from noncarbohydrates (ex: cortisone) | AP Bio Ch. 45 Vocab |
| 11 | glucocorticoids | glucose metabolism hormones that increase the availability of glucose - stimulate gluconeogenesis, promote storage of glycogen; high doses can cause increased glucose in plasma which mimics diabetes; cause protein catabolism which at high doses manifests as thinning of skin and wasting of muscles; causes metabolism of fat, which manifests at high levels as fat redistribution, buffalo hump etc (cortisol) | 351 test 2 |
| 12 | glucocorticoids | influence the metabolism of carbohydrates, proteins, and fats; affect skeletal muscle, the cardiovascular system, and the CNS; at times of stress are essential for survival | 351 test 2 |
| 13 | glucocorticoids | an adjuvant therapy which is used to decrease edema and inflammation, and create a sense of well being and appetitie. ex. prednisone and dexamethasone. | adtn 345 exam 3 |
| 14 | Glucocorticoids | cortisol (natural), prednisone, cortisone, dexamethasone (synthetic). Increase blood sugar levels. Decrease inflammation. Helpful in times of stress to maintain homeostasis. | Anatomy & Physiology: Endocrine System |
| 15 | glucocorticoids | maintains glucose levels | Ch.19.1-Endocrine System |
| 16 | Glucocorticoids | Cortisone, coritisol; raises blood sugar; resists stress | Anatomy and Physiology Ch. 18 - Part 4 |
| 17 | Glucocorticoids | Cortisol "steroids", promotes gluconeogenesis, inhibits immune system | Veterinary Science : Clin. Path.(Final Exam Review) |
| 18 | Glucocorticoids | Cushing's syndrome- moon face, depression of the immune system | Chapter 9- Conditions Caused by Endocrine Organ Hypoactivity or Hyperactivity |
| 19 | Glucocorticoids | Increase blood glucose | Endocrine System |
| 20 | Glucocorticoids | Cortisol, | Clin Path: Endocrine Disorders |
| 21 | Glucocorticoids | Involved in glucose regulation and protein metabolism; important to deal with stress. (Adrenal Cortex) | Endocrine System |
| 22 | Glucocorticoids | used for Addisons dz, Inflammation and immune supression as well as Asthma. INHIBITS PHOSPHOLIPASE A2 AND EXPRESSION OF COX-2decrease prod of Leukotrienes and Prostaglandins (causes cushings like sx and peptic ulcers) | First aid endocrine and repro drugs (no antihyperglycemics) |
| 23 | glucocorticoids | raises blood sugar evels | Ch 15 Endocrine Hormones |
| 24 | Glucocorticoids | decreases the production of leukotrienes and prostaglandins by inhibiting phospholipase A2 and inhibiting the expression of COX-2; used to treat Addison's disease, inflammation, asthma, and induce immune suppression; can result in iatrogenic Cushing's syndrome [Hydrocortisone, Prednisone, Triamcinolone, Dexamethasone, Beclomethasone] | USMLE Pharm |
| 25 | Glucocorticoids | Raises blood sugar levels, Adrenal cortex | Endocrine Hormones |
| 26 | glucocorticoids | a class of steroid hormones, released by the adrenal cortex, that affect carbohydrate metabolism and inflammation | Physio Ch. 5 |
| 27 | Glucocorticoids | Not considered a cure for atopic dermatitis but rather a short duration of therapy only recommended as a last resort type of therapy; associated with a cancer risk. (Corticosteroids/Antihistamines/Glucocorticoids) | Pathophysiology Test 1 |
| 28 | Glucocorticoids | Management of CLL associated thromboctyopenia and autoimmune hemolytic syndrome | Anti-Cancer Drugs |
| 29 | Glucocorticoids | Dexamethasone, Prednisone, Hydrocortisone | Anti-Cancer Drugs |
| 30 | Glucocorticoids | maintain the carbohydrate reserve of the body by stimulating the liver to convert amino acids into glucose instead of protein. | Unit 6 Endocrine |
| 31 | Glucocorticoids | Mech = GCR a=Down, b=up, Bridge Therapy, IA injections, AE: HPA | DMARDs |
| 32 | glucocorticoids | The group of steroid hormones that helps regulate carbohydrate, lipid, and protein metabolism. | MAS 112-Ch. 13 |
| 33 | glucocorticoids | influence glucose metabolism; stimulated by ACTH; anti inflammatory response and suppresses allergic reactions; elevates blood sugar | endocrine pathology |
| 34 | glucocorticoids | anti-inflammatory, regulate carb metabolism | Endocrine System |
| 35 | Glucocorticoids | Targets liver to make glucose from fats and protein which is released into blood | Adrenal Cortex |
| 36 | glucocorticoids | affect protein, fat and glucose metabolism | A&P 2 hormone test |
| 37 | glucocorticoids | adrenal gland-cortex | A&P glands |
| 38 | Glucocorticoids | 1)involved in sugar metabolism and stress, suppresses immune system, CORTAID-suppresses inflammation | endocrinology |
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