| Term | Definition |
|
Intercalated discs |
Thickened area on the membrane surrounding the muscle fiber, where muscle fibers connect to each other |
|
Desmosomes |
Cell junctions that hold fibers tightly together |
|
Gap junctions |
Allows nerve impulses to travel from one fiber to the next |
|
Sinoatrial node (SA node) |
"Pacemaker of the heart"; located in the upper right attrium that receives impulses from the ANS and sends them to the left side of the heart, causing the atria to contract |
|
Atrioventricular node (AV node) |
Located in the lower right attrium; receives impulses from the SA node and sends them to the Bundle of His |
|
Bundle of His |
Sends impulses down the top of the interventricular septum; bundle splits into left and right bundle branches |
|
Perkinje fibers |
End of the conduction system; sends impulses through the ventricles, causing them to contract |
|
Electrocardiogram |
Graphic record of the heart's electrical activity; EKG |
|
P wave |
Represents atrial contraction (depolarization) |
|
QRS complex |
Represents ventricular contraction (depolarization) |
|
T wave |
Represents ventricular relaxation (repolarization) |
|
Cardiac cycle |
Series of events that occur during one heart beat |
|
Atrial systole |
Time when atria are contracting; most blood moves from atria into ventricles passively due to gravity; depolarization of the SA node causes atrial contraction, putting pressure on remain blood inside atria, and causing it to move into the ventricles |
|
Ventricular systole |
Time when ventricles are contracting and atria are relaxed; triggered by ventricular depolarization; as contraction begins, cuspid valve closes and pressure rises in ventricles; once pressure is higher than the exiting vessels, ventricular ejection occurs and blood is forced out through semilunar valves; 70ml of blood exits, while 60ml remains in the ventricle |
|
Relaxation period (diastole) |
Time when both atria and ventricles are relaxed; ventricular repolarization; pressure within the ventricles drops and some blood begins to flow backwards; backflow of blood catches in the semilunar valves, and forces them closed; once ventricular pressure falls below atrial pressure, cuspid valves open |
|
Cardiac output |
Volume of blood ejected from a ventricle each minute; heart rate x stroke volume (CO=HR x SV); value is equal to blood volume at rest |
|
Heart rate |
Times of heart beats per minute |
|
Stroke volume |
Volume of blood ejected by a ventricle with each contraction |
|
Cardiac reserve |
The difference between a person's maximun cardiac output and cardiac output at rest; the average person has a value 4-5 times the resting value; is limited or absent in ppl with severe heart disease |
|
Regulation of stroke volume |
If extra blood enters ventricle, more blood will exit; whatever enters the heart must eventually go out; stroke volume tends to be about 60% of the total volume of blood that enters the ventricles, while 40% remains in the ventricle |
|
Preload |
Degree of stretch on the heart right before contraction; greater stretch on cardiac muscle fibers determines greater strength of contraction |
|
Frank-Starling Law of the heart |
The more the heart fills with blood, the greater the contraction due to the increased amount of stretching |
|
Contractility |
Forcefulness of contraction of the cardiac muscle fibers |
|
Positive inotropic agents |
Increases the strength of heart contraction; e.g. epinephrine (adrenaline), digitalis; sympathetic nervous system is activated |
|
Negative inotropic agents |
Decreases strength of heart contraction; e.g. anesthetics; anoxia (insufficient oxygen); inhibition of sympathetic nervous system |
|
Afterload |
Pressure that must occur in the ventricles to force open the semilunar valves; ejection of blood; blood leaves ventricles when pressure in the right ventricle is greater than in the pulmonary trunk and when pressure in the left ventricle is greater than in aorta |
|
Average heart rate |
75 BPM (resting) |
|
Tachycardia |
High (fast) heart rate greater than 100 BPM |
|
Bradycardia |
Low (slow) heart rate less than 60 BPM |
|
Cardiovascular center |
Located in the medulla oblongata; can increase or decrease nerve impulses that signal the heart |
|
Chemoreceptors |
Receptors that monitor chemical changes in blood; e.g. oxygen and nutrient levels |
|
Baroreceptors |
Monitors stretching of blood vessels caused by blood pressure |
|
Epinephrine |
Hormone that increases HR and contractility |
|
Cations |
Chemicals that control HR; e.g. sodium, potassium, and calcium |
|
Lower HR |
Caused by physical factors such as increased fitness or increased age |
|
Higher HR |
Caused by physical factors such as gender (females), increased body temperature, excercise, or increased body mass |